By Thomas C. Spoor
There are various nice neuro-ophthalmology texts to be had starting from large encyclopedic tomes to small, specified assets. Combining the easiest beneficial properties of those books, Atlas of Neuro-ophthalmology bargains a glimpse right into a wide selection of infrequent and weird neuro-ophthalmic problems. lengthy well-known as a number one authority at the optic nerve, Tom Spoor distills his years of expertise into this beautifully illustrated atlas.
The booklet is a very revised and up-to-date model of Dr. Spoor's unique Atlas of Optic Nerve problems (1992). He addresses all facets of the optic nerve and its disorders-such as optic atrophy, neuropathy, and nerve tumors-making this a vital publication for practitioners and scholars. The inclusion of easy and complicated themes makes it an awesome learn consultant and the wonderful illustrations make it a realistic diagnostic software
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Extra resources for Atlas of neuro-ophthalmology
48). Injury to the optic radiations in the temporal lobe result in a distinctive superior quadrantanopia (Fig. 49), since the inferior fibers pass through the temporal lobe as Meyer’s loop (Fig. 47). Injury to the radiations in the parietal lobe results in an inferior quadrantanopia due to injury to the superior fibers of the optic radiations (Fig. 50). Occipital lobe injury results in very congruous homonymous hemianopias, either involving just central vision (occipital tip lesion) or the entire hemifield (Fig.
Can J Ophthalmol 1996; 31:311–13. 8. Moster ML, Galetta SL, Schatz NJ. Physiologic ‘functional’ visual loss. Surv Ophthalmol 1996; 40:395–9. 9. Thompson HS. Functional visual loss. Am J Ophthalmol 1985; 100:209–13. 10. Keltner JL. The California syndrome: a threat to all. Arch Ophthalmol 1988; 106:1053–4. Chapter 3 The optic nerve ANATOMY Each optic nerve is composed of approximately one million axons from retinal ganglion cells. These axons exit the eye through the lamina cribrosa—where they are most susceptible to damage from either elevated intraocular or intracranial pressure—intermingle with axons from the opposite eye to form the optic chiasm and optic tracts, and finally synapse at the lateral geniculate bodies.
31). All other optic atrophy requires further evaluation to determine its etiology. Neuroimaging is readily available and should be utilized if the cause of the optic atrophy is not obvious. 27 Optic atrophy due to obvious choroidal tears in patient suffering blunt trauma to the eye. 28 Attenuated arterioles and optic atrophy in a patient with an antecedent CRAO.