Endocrinology of Cardiovascular Function by Gabor M. Rubanyi M.D., Ph.D. (auth.), Ellis R. Levin M.D.,

By Gabor M. Rubanyi M.D., Ph.D. (auth.), Ellis R. Levin M.D., Jerry L. Nadler M.D. (eds.)

Endocrinology of Cardiovascular Function is a becoming inauguration to the Endocrine replace Series. the purpose of those courses is to supply the clinician with innovative, but succinct, entry to the most recent advances in endocrinology. traditionally, our figuring out of hormonal disturbances was once limited to the classical secretory glands and their ambitions. As Endocrinology of Cardiovascular Function so aptly exhibits, endocrinology is not any longer limited through our early physiologic knowing of glandular disease. Endocrinology ofCardiovascular Function has set the normal of excellence for the long run volumes during this sequence.
Shlomo Melmed, M.D. sequence Editor, Endocrine Update
progress elements equivalent to IGF-1 play very important roles in cardiovascular mobile hypertrophy and the reaction to acute vascular damage. From one other point of view, conventional endocrine hormones, akin to estrogen, were discovered to take part in fighting the advance of atherosclerosis in girls, appearing via novel mechanisms on track vascular cells. different `endocrine' hormones, equivalent to PTHRP and adrenomedullin, additionally modulate cardiovascular and renovascular dynamic states, suggesting new roles for those peptides as vasodilators. This multi-authored textual content is devoted to highlighting rising and critical new information about the endocrinology of the cardiovascular approach.
Ellis R. Levin, M.D.

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266(28): 19034-19039, 1991. 49. : Transcriptional regulation of the human cholecystokinin gene: composite action of upstream stimulatory factor, Spl, and members of the CREB/ATF-AP-I family of transcription factors. DNA and Cell Biology. 15(1): 53-63, 1996. 50. F. : Estrogen inhibits the vascular injury response in estrogen receptor alpha-deficient mice. Nature Medicine. 3(5): 545-548, 1997. 43 51. : endothelium-derived nitric oxide production in the mouse aorta. Journal of Clinical Investigation.

204ET ConI. o Relative CAT Activity ". j:>. Vol 35 Figure 2-Effect of estradiol (E) or progesterone (P) on stimulated ET-I transcription in BAEC, as determined by transfected CAT reporter constructs. Individual dishes of BAEC were transiently transfected and relative CAT activity was calculated. M. from at least 3 experiments combined, each condition in triplicate/experiment. 043ET-lICAT reporter. 109. 143 ET-I CAT activity. (C) All sitmulation of ET-I transcription is inhibited by estradiol (E) or progesterone (P).

Effects of estradiol and progesterone on the increased synthesis of collagen in atherosclerotic rabbit aortas. Atherosclerosis. 54(2): 177-185, 1985. : Effects of hormone-replacement therapy on fibrinolysis in postmenopausal women. The New England Journal of Medicine. 336(10): 683-690, 1997. : Plasminogen activator inhibitors. Blood. 69: 381387, 1987. : Association between 41 17. 18. 19. 20. 21. 22. 23. 24. 25. 26. 27. 28. 29. 30. 31. 32. 33. increased estrogen status and increased fibrinolytic potential in the Framingham Offspring Study.

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